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TREM-2, Monoclonal Antibody

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產(chǎn)品名稱: TREM-2, Monoclonal Antibody
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TREM-2, Monoclonal Antibody


TREM-2, Monoclonal Antibody  的詳細(xì)介紹
Product Name

TREM-2, Monoclonal Antibody

Full Product Name

TREM-2, Mouse, mAb 6E9, FITC

Product Synonym Names
TREM-2, Mouse, clone 6E9, FITC; Triggering receptor expressed on monocytes 2; TREM2
Product Gene Name

anti-TREM-2 antibody

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Research Use Only
For Research Use Only. Not for use in diagnostic procedures.
TOP
OMIM
221770
3D Structure
ModBase 3D Structure for Q9NZC2
Clonality
Monoclonal
Isotype
Rat IgG2b
Clone Number
6E9
Host
Rat
Species Reactivity
Mouse
Form/Format
1 ml (100 ug/ml) 0.2 um filtered antibody solution in PBS, containing 0.1% bovine serum albumin and 0.02% sodium azide.
Immunogen
Mouse recombinant TREM-2 protein
Conjugate
FITC
Preparation and Storage
Product should be stored at 4 degree C. Under recommended storage conditions, product is stable for one year.
Other Notes
Small volumes of anti-TREM-2 antibody vial(s) may occasionally become entrapped in the seal of the product vial during shipment and storage. If necessary, briefly centrifuge the vial on a tabletop centrifuge to dislodge any liquid in the container`s cap. Certain products may require to ship with dry ice and additional dry ice fee may apply.
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Related Product Information for
anti-TREM-2 antibody
The monoclonal antibody 6E9 recognizes mouse membrane-bound as well as soluble triggering receptor expressed on myeloid cells-2 (TREM-2). TREM-2 is a 26 kDa transmembrane glycoprotein that consists of a single extracellular immunoglobulin-like domain, a transmembrane region with a charged lysine residue and a short cytoplasmic tail. It associates with DNAX-activation protein 12 (DAP12) for signaling and function. TREM-2 is expressed on immature monocyte-derived dendritic cells. After activation by microbial products or tumor necrosis factor (TNF) and TNF-related proteins, dendritic cells downregulate the expression of TREM-2. TREM-2 is also expressed by osteoclasts and microglia, where it is involved in bone modeling and brain function, respectively. Another role of TREM-2 might be promoting the removal of apoptotic cells, organic matrix and macromolecules by microglia. Defects in TREM-2 are a cause of polycystic lipomembranous osteodysplasia with sclerosing leukoencephalopathy (PLOSL), also called presenile dementia with bone cysts or Nasu-Hakola disease (NHD). TREM-2, like TREM-1, can be cleaved on the membrane to release a soluble form of TREM-2 (sTREM-2). Elevated levels of sTREM-2 in CSF of multiple sclerosis patients have been detected. This elevated level may inhibit the anti-inflammatory function of the membrane-bound receptor suggesting sTREM-2 to be a possible target for future therapies.
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